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A further phenomenon, observed by this study and that of Fründt et al. (2017) is that of DMA. Both studies are the first to experimentally measure DMA in ASD, observing this in a subset of the ASD groups. DMA is the experience of perceiving innocuous touch, such as gentle stroking, as aversive, a phenomenon observed in ASD sensory over-responsivity literature (Baranek and Berkson 1994; Green et al. 2015; Reynolds and Lane 2008). Central sensitisation i.e. changes in signalling in the spinal cord (Campbell and Meyer 2006), is commonly thought to underlie DMA (Gierthmühlen et al. 2012), as it is the increased response of neurons to stroking stimuli. Intriguingly, some groups have offered a peripheral explanation for DMA (Liljencrantz et al. 2013), whereby an alteration in C-tactile afferent function, which typically mediates a pleasant percept associated with low force slow stroking touch, communicates noxious experience. This explanation then lends weight to research suggesting that an early mechanism behind ASD may be an alteration in CT fibre function (Cascio et al. 2018; Gordon et al. 2013; Kaiser et al. 2016; Walker and McGlone 2013). It is clear that this proposition requires further investigation. However, QST cannot fully distinguish between central and peripheral alterations (Mücke et al. 2014), therefore we can only speculate at this time. 781b155fdc